2959 7 13912 1 36669 document 13912 accustic stimulation km ia.pdf application/pdf df03f8c550197161e46ac551efbd1ff4 MD5 195826 2015-06-25 11:46:34 http://eprints.iliauni.edu.ge/2959/1/accustic%20stimulation%20km%20ia.pdf 2959 1 1 application/pdf text en public

accustic stimulation km ia.pdf

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http://eprints.org/relation/isVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/isVolatileVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/ismediumThumbnailVersionOf http://eprints.iliauni.edu.ge/id/document/13912 13916 2 36677 document 13916 small.jpg image/png e89200b50971bdceb6699e05f55f397c MD5 1903 2015-06-25 11:56:48 http://eprints.iliauni.edu.ge/2959/5/small.jpg 2959 5 5 image/png other Thumbnails conversion from text to thumbnail_small ge public

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http://eprints.org/relation/isVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/isVolatileVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/issmallThumbnailVersionOf http://eprints.iliauni.edu.ge/id/document/13912 30622 1 71461 document 30622 indexcodes.txt text/plain 901569c14764d4cc8b7dec63cdd7244f MD5 3957 2016-07-25 14:14:01 http://eprints.iliauni.edu.ge/2959/6/indexcodes.txt 2959 6 6 text/plain other Generate index codes conversion from text to indexcodes ge public

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http://eprints.org/relation/isVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/isVolatileVersionOf http://eprints.iliauni.edu.ge/id/document/13912 http://eprints.org/relation/isIndexCodesVersionOf http://eprints.iliauni.edu.ge/id/document/13912 archive 87 disk0/00/00/29/59 2015-06-26 05:50:45 2015-06-26 05:50:45 2015-06-26 05:50:45 article show Zhvania Mzia G mzia_zhvania@iliauni.edu.ge Dzhaparidze Nadezhda nafia_japaridze@yahoo.com Bolkvadze Tamar t_bolkvadze@yahoo.com Kotariya Nato n_kotaria@yahoo.com pub Neurobiology ins10 public Krushinskii—Molodkina rats are genetically predisposed to the development of seizure activity in response to acoustic stimulation. These animals are a natural model to study the mechanisms of hereditary and audiogenic epilepsy. The main cause of these disorders is a deficiency of GABAergic transmission [3-5]. Biochemical and electrophysiological studies showed that this deficiency accompanies the increase in excitation transmission and is most pronounced in auditory structures, particularly in the relay structure (inferior colliculi of the quadrigeminal plate) [4-6]. The number of basket and GABA-containing neurons is maximum in this structure. The presence of a considerable number of these cells should contribute to the formation of the epileptic focus [5,7]. It was hypothesized that rats with genetic predisposition to audiogenic seizures differ from healthy animals in the structure of GABAAergic receptors in auditory structures [5,6]. It remains unknown whether seizure activity induced by epileptogenic acoustic stimulation can cause structural changes in auditory brain structures of these rats. Neuronal death is a typical morphological sign of various forms of epilepsy. Quantitative analysis of main cells (interneurons and GABA-containing cells) in the central nucleus of the inferior colliculi and anterior and posterior regions of the auditory cortex in Krushinskii—Molodkin rats was performed 1 month after audiogenic epileptic activity induced by acoustic stimulation. 2006 published Bulletin of Experimental Biology and Medicine 142 4 Springer, Translated from the Journal of Russian Academy of Science 419-421 TRUE